Early and Sustained Activation of Autophagy in Degenerating Axons after Spinal Cord Injury-Reference-Cited by-同舟云学术

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Early and Sustained Activation of Autophagy in Degenerating Axons after Spinal Cord Injury

Published:2014-09-12 Issue:2 Volume:25 Page:157-170
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:Brain Pathology

Author:

Ribas Vinicius Toledo¹,Schnepf Bianca¹,Challagundla Malleswari¹,Koch Jan Christoph¹,Bähr Mathias¹²,Lingor Paul¹²

Affiliation:

1. Department of Neurology; University Medicine Göttingen; Göttingen Germany

2. Center for Nanoscale Microscopy and Molecular Physiology of the Brain (CNMPB); Göttingen Germany

Funder

International Foundation for Research in Paraplegia

Deutsche Forschungsgemeinschaft

Center for Nanoscale Microscopy and Molecular Physiology of the Brain

Publisher

Wiley

Subject

Clinical Neurology,Pathology and Forensic Medicine,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bpa.12170/fullpdf

Reference37 articles.

1. Sirna screening of the kinome identifies ULK1 as a multidomain modulator of autophagy;Chan;J Biol Chem,2007

2. Autophagy is activated in injured neurons and inhibited by methylprednisolone after experimental spinal cord injury;Chen;Spine (Phila. PA. 1976),2012

3. Akt suppresses retrograde degeneration of dopaminergic axons by inhibition of macroautophagy;Cheng;J Neurosci,2011

4. The atg8 and atg12 ubiquitin-like conjugation systems in macroautophagy. “Protein modifications: beyond the usual suspects” review series;Geng;EMBO Rep,2008

5. Withdrawal of essential amino acids increases autophagy by a pathway involving Ca2+/calmodulin− dependent kinase kinase- (CaMKK-);Ghislat;J Biol Chem,2012

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