Cx43 and AKAP95 regulate G1 /S conversion by competitively binding to cyclin E1 / E2 in lung cancer cells-Reference-Cited by-同舟云学术

Cx43 and AKAP95 regulate G1 /S conversion by competitively binding to cyclin E1 / E2 in lung cancer cells

Published:2020-04-27 Issue:6 Volume:11 Page:1594-1602
ISSN:1759-7706
Container-title:Thoracic Cancer
language:en
Short-container-title:Thorac Cancer

Author:

Chen Renzhen¹,Chen Yu²,Yuan Yangyang³,Zou Xuan¹,Sun Qian¹,Lin Hongyan¹,Chen Xiaoyi¹,Liu Mingda¹,Deng Zifeng¹,Yao Youliang¹,Guo Dongbei¹,Zhang Yongxing¹

Affiliation:

1. State Key Laboratory of Molecular Vaccinology and Molecular Diagnostics, School of Public HealthXiamen University Xiamen China

2. School of MedicineXiamen University Xiamen China

3. Henan provincial Clinical Research Center for Perinatal MedicineThe Third Affiliated Hospital of Zhengzhou University Zhengzhou China

Funder

The Autonomous Research Fund of State Key Laboratory of Molecular Vaccinology and Molecular Diagnosticsh

The Education Scientific Research Project of Young Teachers of 8 Fujian Province

The National Natural Science Foundation of China

The Natural Science Foundation of Fujian Province of China

Publisher

Wiley

Subject

Pulmonary and Respiratory Medicine,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1759-7714.13435

Reference31 articles.

1. Dynamic changes in protein interaction between AKAP95 and Cx43 during cell cycle progression of A549 cells

2. A-Kinase-Anchoring Protein 95 Functions as a Potential Carrier for the Nuclear Translocation of Active Caspase 3 through an Enzyme-Substrate-Like Association

3. Enhanced connexin 43 expression delays intra-mitoitc duration and cell cycle traverse independently of gap junction channel function

4. Connexin 43 hemichannels contribute to the propagation of apoptotic cell death in a rat C6 glioma cell model

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