Cyclosporin A-induced oxidative stress is not the consequence of an increase in mitochondrial membrane potential-Reference-Cited by-同舟云学术

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Cyclosporin A-induced oxidative stress is not the consequence of an increase in mitochondrial membrane potential

Published:2007-05-17 Issue:12 Volume:274 Page:3003-3012
ISSN:1742-464X
Container-title:FEBS Journal
language:en
Short-container-title:

Author:

van der Toorn Marco,Kauffman Henk F.,van der Deen Margaretha,Slebos Dirk-Jan,Koëter Gerard H.,Gans Rijk O. B.,Bakker Stephan J. L.

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1742-4658.2007.05827.x/fullpdf

Reference39 articles.

1. Cellular and molecular mechanisms of cyclosporin nephrotoxicity;Kopp;J Am Soc Nephrol,1990

2. Oxidative and nitrosative stress in kidney disease: a case for cyclosporine A;Redondo-Horcajo;J Nephrol,2005

3. Cyclosporin A blocks muscle differentiation by inducing oxidative stress and inhibiting the peptidyl-prolyl-cis-trans isomerase activity of cyclophilin A: cyclophilin A protects myoblasts from cyclosporin A-induced cytotoxicity;Hong;FASEB J,2002

4. Does cyclosporin A generate free radicals?;Buetler;Trends Pharmacol Sci,2000

5. Cyclosporin A-induced hydrogen peroxide synthesis by cultured human mesangial cells is blocked by exogenous antioxidants;Perez de Lema;Life Sci,1998

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