Suppression of NADPH oxidase 2 substantially restores glucose-induced dysfunction of pancreatic NIT-1 cells
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1742-4658.2010.07911.x/fullpdf
Reference20 articles.
1. Thioredoxin-interacting protein: a critical link between glucose toxicity and β-cell apoptosis;Chen;Diabetes,2008
2. Glucolipotoxicity: fuel excess and β-cell dysfunction;Poitout;Endocr Rev,2008
3. Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity;Newsholme;J Physiol,2007
4. Evidence for a role of superoxide generation in glucose-induced β-cell dysfunction in vivo;Tang;Diabetes,2007
5. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology;Bedard;Physiol Rev,2007
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