cMet agonistic antibody attenuates apoptosis in ischaemia‐reperfusion–induced kidney injury-Reference-Cited by-同舟云学术

cMet agonistic antibody attenuates apoptosis in ischaemia‐reperfusion–induced kidney injury

Published:2020-04-02 Issue:10 Volume:24 Page:5640-5651
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

An Jung Nam¹,Li Lilin²³,Lee Junghun⁴,Yu Seung‐Shin⁴,Lee Jeonghwan⁵,Kim Yong Chul⁶,Kim Dong Ki²⁶,Oh Yun Kyu²⁵,Lim Chun Soo²⁵,Kim Yon Su²⁶,Kim Sunyoung⁴,Yang Seung Hee⁷⁸^ORCID,Lee Jung Pyo²⁵^ORCID

Affiliation:

1. Department of Internal Medicine Hallym University Sacred Heart Hospital Anyang Korea

2. Department of Internal Medicine Seoul National University College of Medicine Seoul Korea

3. Department of Intensive Care Unit Yanbian University Hospital Jilin China

4. R&D Center for Innovative Medicines Helixmith Co., Ltd. Seoul Korea

5. Department of Internal Medicine Seoul National University Boramae Medical Center Seoul Korea

6. Department of Internal Medicine Seoul National University Hospital Seoul Korea

7. Kidney Research Institute Seoul National University Seoul Korea

8. Biomedical Research Institute Seoul National University Hospital Seoul Korea

Funder

clinical research grant-in-aid from the Seoul National University Boramae Medical Center

Korea Healthcare Technology R&D Project, Ministry for Health and Welfare, Republic of Korea

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.15225

Reference29 articles.

1. Chronic Kidney Disease After Acute Kidney Injury Requiring Continuous Renal Replacement Therapy and Its Impact on Long-Term Outcomes

2. Sulfatide-Reactive Natural Killer T Cells Abrogate Ischemia-Reperfusion Injury

3. Transcriptional modulation of the T helper 17/interleukin 17 axis ameliorates renal ischemia-reperfusion injury

4. Periostin induces kidney fibrosis after acute kidney injury via the p38 MAPK pathway

5. Soluble Epoxide Hydrolase Activity Determines the Severity of Ischemia-Reperfusion Injury in Kidney

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