Mutations in the lamin B1 gene are not present in multiple sclerosis-Reference-Cited by-同舟云学术

Mutations in the lamin B1 gene are not present in multiple sclerosis

Published:2009-04 Issue:4 Volume:16 Page:544-546
ISSN:1351-5101
Container-title:European Journal of Neurology
language:en
Short-container-title:

Author:

Brussino A.,D’Alfonso S.,Cagnoli C.,Di Gregorio E.,Barberis M.,Padovan S.,Vaula G.,Pinessi L.,Squadrone S.,Abete M. C.,Collimedaglia L.,Guerini F. R.,Migone N.,Brusco A.

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1468-1331.2009.02536.x/fullpdf

Reference12 articles.

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2. Lame ducks or fierce creatures? The role of oligodendrocytes in multiple sclerosis;Zeis;Journal of Molecular Neuroscience,2008

3. Primary-progressive multiple sclerosis;Miller;Lancet Neurology,2007

4. Hereditary adult-onset leukodystrophy simulating chronic progressive multiple sclerosis;Eldridge;New England Journal of Medicine,1984

5. Lamin B1 duplications cause autosomal dominant leukodystrophy;Padiath;Nature Genetics,2006

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1. A longitudinal study of a family with adult-onset autosomal dominant leukodystrophy: Clinical, autonomic and neuropsychological findings;Autonomic Neuroscience;2016-02

2. Lamin B1 overexpression increases nuclear rigidity in autosomal dominant leukodystrophy fibroblasts;The FASEB Journal;2014-05-22

3. Neurology in theEuropean Journal of Neurology;European Journal of Neurology;2010-11-18

4. Autosomal Dominant Leukodystrophy Caused by Lamin B1 Duplications: A Clinical and Molecular Case Study of Altered Nuclear Function and Disease;Methods in Cell Biology;2010