Increased GABA synthesis in skin fibroblasts of patients with Huntington's disease: a possible role of glutamic acid decarboxylase?-Reference-Cited by-同舟云学术

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Increased GABA synthesis in skin fibroblasts of patients with Huntington's disease: a possible role of glutamic acid decarboxylase?

Published:1996-09 Issue:5 Volume:3 Page:467-470
ISSN:1351-5101
Container-title:European Journal of Neurology
language:en
Short-container-title:

Author:

Gerlach M.,Kuhn W.,Müller Th.,Winkel R.,Lange H.,Przuntek H.

Publisher

Wiley

Subject

Clinical Neurology,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1468-1331.1996.tb00251.x/fullpdf

Reference24 articles.

1. Huntington's disease and Tourette syndrome;Comings;American Journal of Human Genetics,1981

2. Biochemische Arbeitsmethoden

3. Targeting of the 67-kDa isoform of glutamic acid decarboxylase to intracellular organelles is mediated by ist interaction with the NH2-terminal region of the 65-kDa isoform of glutamic acid decarboxylase;Dirkx;Journal of Biological Chemistry,1995

4. A simple, sensitive and specific radioreceptor assay for endogenous GABA in brain tissue;Enna;Journal of Neurochemistry,1976

5. Gamma-Aminobutyric acid (GABA) in human cerebrospinal fluid: radioreceptor assay;Enna;Journal of Neurochemistry,1977

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