Kallistatin prevents ovarian hyperstimulation syndrome by regulating vascular leakage-Reference-Cited by-同舟云学术

Kallistatin prevents ovarian hyperstimulation syndrome by regulating vascular leakage

Published:2022-07-21 Issue:16 Volume:26 Page:4613-4623
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cellular Molecular Medi

Author:

Huang Jianfang¹²,Mao Yuling¹,Li Quanxin³,Hong Honghai⁴,Tang Ni¹,Kang Xiangjin¹,Huang Yuling¹,Liu Jianqiao¹,Gong Qing³,Yao Yachao⁵,Li Lei¹⁶^ORCID

Affiliation:

1. Key Laboratory for Major Obstetric Diseases of Guangdong Province Department of Obstetrics and Gynecology, Center for Reproductive Medicine The Third Affiliated Hospital of Guangzhou Medical University Guangzhou China

2. Department of Reproductive Medicine Ganzhou People's Hospital Ganzhou Jiangxi China

3. Department of Biochemistry and Molecular Biology, GMU‐GIBH Joint School of Life Sciences Guangzhou Medical University Guangzhou China

4. Department of Clinical Laboratory The Third Affiliated Hospital of Guangzhou Medical University Guangzhou China

5. Department of Laboratory Medicine Guangdong Second Provincial General Hospital Guangzhou China

6. Key Laboratory for Reproductive Medicine of Guangdong Province The Third Affiliated Hospital of Guangzhou Medical University Guangzhou China

Funder

National Nature Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.17491

Reference43 articles.

1. Neurovascular Complications of Ovarian Hyperstimulation Syndrome (OHSS): From Pathophysiology to Recent Treatment Options

2. Ovarian hyperstimulation syndrome

3. Vascular endothelial growth factor as capillary permeability agent in ovarian hyperstimulation syndrome

4. Human chorionic gonadotropin-dependent expression of vascular endothelial growth factor/vascular permeability factor in human granulosa cells: importance in ovarian hyperstimulation syndrome

5. Ovarian hyperstimulation syndrome is correlated with a reduction of soluble VEGF receptor protein level and a higher amount of VEGF-A

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