Tumour necrosis factor-αcontributes to improved cardiac ischaemic tolerance in rats adapted to chronic continuous hypoxia

Author:

Chytilová A.12,Borchert G. H.1,Mandíková-Alánová P.13,Hlaváčková M.14,Kopkan L.3,Khan Md. A. Hye5,Imig J. D.5,Kolář F.1,Neckář J.13

Affiliation:

1. Institute of Physiology; The Czech Academy of Sciences; Prague Czech Republic

2. Department of Physiology; Faculty of Science; Charles University in Prague; Prague Czech Republic

3. Center for Experimental Medicine; Institute for Clinical and Experimental Medicine; Prague Czech Republic

4. Department of Cell Biology; Faculty of Science; Charles University in Prague; Prague Czech Republic

5. Department of Pharmacology & Toxicology; Medical College of Wisconsin; Milwaukee WI USA

Funder

Czech Science Foundation

European Commission within the Operational Program Prague–Competitiveness

Grant Agency of the Charles University in Prague

Publisher

Wiley

Subject

Physiology

Reference57 articles.

1. Effect of adaptation to intermittent high altitude hypoxia on ischemic ventricular arrhythmias in rats;Asemu;Physiol Res,2000

2. N-acetylcysteine treatment prevents the up-regulation of MnSOD in chronically hypoxic rat hearts;Balková;Physiol Res,2011

3. The late phase of preconditioning;Bolli;Circ Res,2000

4. Mitochondrial BKCa channels contribute to protection of cardiomyocytes isolated from chronically hypoxic rats;Borchert;Am J Physiol Heart Circ Physiol,2011

5. Eccentric cardiac hypertrophy was induced by long-term intermittent hypoxia in rats;Chen;Exp Physiol,2007

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