Substantial depletion of the intracellular Ca2+stores is required for macroscopic activation of the Ca2+release‐activated Ca2+current in rat basophilic leukaemia cells
Author:
Affiliation:
1. Laboratory of Molecular ar 11:and1:Cellular Signalling, Department of Physiology, University of Oxford, Parks Road, Oxford OX1 3PT, UK
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1469-7793.2000.t01-1-00247.x
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2. Antigen and thapsigargin promote influx of Ca2+ in rat basophilic RBL-2H3 cells by ostensibly similar mechanisms that allow filling of inositol 1,4,5-trisphosphate-sensitive and mitochondrial Ca2+ stores
3. Ca2+-dependent capacitance increases in rat basophilic leukemia cells following activation of store-operated Ca2+ entry and dialysis with high-Ca2+-containing intracellular solution
4. Inositol trisphosphate and calcium signalling
5. Simultaneous measurements of Ca2+in the intracellular stores and the cytosol of hepatocytes during hormone-induced Ca2+oscillations
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