Inhibition of KCNQ1‐4 potassium channels expressed in mammalian cells via M 1 muscarinic acetylcholine receptors-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Inhibition of KCNQ1‐4 potassium channels expressed in mammalian cells via M 1 muscarinic acetylcholine receptors

Published:2000-02 Issue:3 Volume:522 Page:349-355
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Selyanko A. A.¹,Hadley J. K.¹,Wood I. C.²,Abogadie F. C.²,Jentsch T. J.³,Brown D. A.¹

Affiliation:

1. Department of Pharmacology, University College London, Gower Street, London WC1E 6BT, UK

2. Wellcome Laboratory for Molecular Pharmacology, University College London, Gower Street, London WC1E 6BT, UK

3. Zentrum für Molekulare Neurobiologie Hamburg (ZMNH), Universität Hamburg, Martinistrasse 85, D‐20246 Hamburg, Germany

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1469-7793.2000.t01-2-00349.x

Reference28 articles.

1. KvLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current

2. Characterization of muscarinic receptor subtypes inhibiting Ca2+ current and M current in rat sympathetic neurons.

3. A Potassium Channel Mutation in Neonatal Human Epilepsy

5. A pore mutation in a novel KQT-like potassium channel gene in an idiopathic epilepsy family

Cited by 146 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. KCNQ2/3 regulates efferent mediated slow excitation of vestibular afferents in mammals;2024-01-01

2. Cholinergic Stimulation Modulates the Functional Composition of CA3 Cell Types in the Hippocampus;The Journal of Neuroscience;2023-06-05

3. The second PI(3,5)P₂ binding site in the S0 helix of KCNQ1 stabilizes PIP₂-at the primary PI1 site with potential consequences on intermediate-to-open state transition;Biological Chemistry;2023-02-23

4. Regulation of Neurotransmitter Release by K+ Channels;Advances in Neurobiology;2023

5. Emerging mechanisms involving brain Kv7 channel in the pathogenesis of hypertension;Biochemical Pharmacology;2022-12

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3