Voltage-dependent inactivation of the human K+channel KvLQT1 is eliminated by association with minimal K+channel (minK) subunits
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1469-7793.1998.037bz.x/fullpdf
Reference22 articles.
1. KvLQT1 and IsK (minK) proteins associate to form the IKs cardiac potassium channel;Barhanin;Nature,1996
2. Tetraethylammonium blockade distinguishes two inactivation mechanisms in voltage-activated K+ channels;Choi;Proceedings of the National Academy of Sciences of the USA,1991
3. Oogenesis in Xenopus laevis (Daudin);Dumont;Journal of Morphology,1972
4. Biophysical and molecular mechanisms of Shaker potassium channel inactivation;Hoshi;Science,1990
5. Two types of inactivation in Shaker K+ channels: effects of alterations in the carboxy-terminal region;Hoshi;Neuron,1991
Cited by 150 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Novel KCNQ1 Q234K variant, identified in patients with long QT syndrome and epileptiform activity, induces both gain- and loss-of-function of slowly activating delayed rectifier potassium currents;Frontiers in Physiology;2024-07-19
2. Inhibitory Perturbations of Fluvastatin on Afterhyperpolarization Current, Erg-mediated K+ Current, and Hyperpolarization-activated Cation Current in Both Pituitary GH3 Cells and Primary Embryonic Mouse Cortical Neurons;Neuroscience;2023-11
3. Exploring single nucleotide polymorphisms in the KCNQ1 gene associated with cardiac disorders;2023-06-26
4. Sensing its own permeant ion: KCNQ1 channel inhibition by external K+;Journal of General Physiology;2023-03-24
5. The second PI(3,5)P2 binding site in the S0 helix of KCNQ1 stabilizes PIP2-at the primary PI1 site with potential consequences on intermediate-to-open state transition;Biological Chemistry;2023-02-23
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3