Dibasic protein kinase A sites regulate bursting rate and nucleotide sensitivity of the cystic fibrosis transmembrane conductance regulator chloride channel
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1469-7793.1998.365bq.x/fullpdf
Reference40 articles.
1. Nucleoside triphosphates are required to open the CFTR chloride channel;Anderson;Cell,1991
2. Coupling of CFTR Cl− channel gating to an ATP hydrolysis cycle;Baukrowitz;Neuron,1994
3. Phosphatase inhibitors activate normal and defective CFTR chloride channels;Becq;Proceedings of the National Academy of Sciences of the USA,1994
4. Regulation of the cystic fibrosis transmembrane conductance regulator Cl− channel by specific protein kinases and phosphatases;Berger;Journal of Biological Chemistry,1993
5. The two nucleotide-binding domains of CFTR have distinct functions in controlling channel activity;Carson;Journal of Biological Chemistry,1995
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