Inositol 1,3,4,5‐tetrakisphosphate enhances long‐term potentiation by regulating Ca 2+ entry in rat hippocampus-Reference-Cited by-同舟云学术

Inositol 1,3,4,5‐tetrakisphosphate enhances long‐term potentiation by regulating Ca 2+ entry in rat hippocampus

Published:1999-05 Issue:3 Volume:516 Page:855-868
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Szinyei Csaba¹,Behnisch Thomas¹,Reiser Georg²,Reymann Klaus G.¹

Affiliation:

1. Project Group Neuropharmacology, Leibniz Institute for Neurobiology, POB 1860, D‐39008 Magdeburg, Germany

2. Institute for Neurobiochemistry, Otto‐von‐Guericke University, Leipziger Straße 44, D‐39120 Magdeburg, Germany

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1469-7793.1999.0855u.x

Reference40 articles.

1. Identification of rat brain p42IP4, a high-affinity inositol(1,3,4,5)tetrakisphosphate/phosphatidylinositol(3,4,5)trisphosphate binding protein

2. Characterization of Ca2+ signals induced in hippocampal CA1 neurones by the synaptic activation of NMDA receptors.

3. Role for ionotropic and metabotropic receptors in quisqualate‐stimulated inositol polyphosphate accumulation in rat cerebral cortex;Baird J. G.;Molecular Pharmacology,1991

4. Induction of LTP in the hippocampus needs synaptic activation of glutamate metabotropic receptors

5. 2,3-Diphosphoglyceric acid blocks long-term potentiation of excitatory postsynaptic currents in hippocampal CA1 neurons of the rat

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