Na/H exchange and H-K ATPase increase distal tubule acidification in chronic alkalosis-Reference-Cited by-同舟云学术

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Na/H exchange and H-K ATPase increase distal tubule acidification in chronic alkalosis

Published:1998-04 Issue:4 Volume:53 Page:945-951
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Wesson Donald E.

Publisher

Elsevier BV

Subject

Nephrology

Reference29 articles.

1. Augmented bicarbonate reabsorption by both the proximal and distal nephron maintains chloride-deplete metabolic alkalosis in rats;Wesson;J Clin Invest,1989

2. Depressed distal tubule acidification corrects chloride-deplete alkalosis in rats;Wesson;Am J Physiol,1990

3. Augmented bidirectional HCO3 transport by rat distal tubules in chronic alkalosis;Wesson;Am J Physiol,1991

4. Maximal proton secretory rate of rat distal tubules is higher during chronic metabolic alkalosis;Wesson;Am J Physiol,1991

5. Renal bicarbonate reabsorption in the rat. IV. Bicarbonate transport mechanisms in the early and late distal tubule;Wang;J Clin Invest,1993

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1. Renal Tubular Acidosis: H+/Base and Ammonia Transport Abnormalities and Clinical Syndromes;Advances in Chronic Kidney Disease;2018-07

2. Molecular Mechanisms and Regulation of Urinary Acidification;Comprehensive Physiology;2014-09-30

3. Clinical Syndromes of Metabolic Alkalosis;Seldin and Giebisch's The Kidney;2013

4. Cellular Mechanisms of Renal Tubular Acidification;Seldin and Giebisch's The Kidney;2013

5. Disorders of Acid-Base Balance;Brenner and Rector's The Kidney;2012

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