Autophagy regulates the localization and degradation of p16 INK4a-Reference-Cited by-同舟云学术

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Autophagy regulates the localization and degradation of p16 INK4a

Published:2020-07 Issue:7 Volume:19 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Coryell Philip R.¹^ORCID,Goraya Supreet K.¹,Griffin Katherine A.¹,Redick Margaret A.¹^ORCID,Sisk Samuel R.¹,Purvis Jeremy E.¹²³⁴

Affiliation:

1. Department of Genetics University of North Carolina at Chapel Hill Chapel Hill North Carolina USA

2. Curriculum for Bioinformatics and Computational Biology University of North Carolina at Chapel Hill Chapel Hill North Carolina USA

3. Lineberger Comprehensive Cancer Center University of North Carolina at Chapel Hill Chapel Hill North Carolina USA

4. Computational Medicine Program University of North Carolina at Chapel Hill Chapel Hill North Carolina USA

Funder

W. M. Keck Foundation

National Institutes of Health

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13171

Reference25 articles.

1. Cytoplasmic, but not nuclear, p16 expression may signal poor prognosis in high-grade astrocytomas

2. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders

3. The Tumor Suppressor Protein p16 and the Human Papillomavirus Oncoprotein-58 E7 Are Naturally Occurring Lysine-less Proteins That Are Degraded by the Ubiquitin System

4. Chapter 12 Monitoring Autophagic Degradation of p62/SQSTM1

5. The crucial impact of lysosomes in aging and longevity

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