LRRK2 recruitment, activity, and function in organelles-Reference-Cited by-同舟云学术

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LRRK2 recruitment, activity, and function in organelles

Published:2021-07-14 Issue: Volume: Page:
ISSN:1742-464X
Container-title:The FEBS Journal
language:en
Short-container-title:FEBS J

Author:

Bonet‐Ponce Luis¹,Cookson Mark R.¹^ORCID

Affiliation:

1. Cell Biology and Gene Expression Section National Institute on Aging National Institutes of Health Bethesda MD USA

Funder

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/febs.16099

Reference148 articles.

1. Cloning of the gene containing mutations that cause PARK8‐linked Parkinson’s disease;Paisán‐Ruíz C;Neuron,2004

2. Mutations in LRRK2 cause autosomal‐dominant parkinsonism with pleomorphic pathology;Zimprich A;Neuron,2004

3. Genome‐wide association study reveals genetic risk underlying Parkinson’s disease;Simón‐Sánchez J;Nat Genet,2009

4. Identification of novel risk loci, causal insights, and heritable risk for Parkinson’s disease: a meta‐analysis of genome‐wide association studies;Nalls MA;Lancet Neurol,2019

5. Large‐scale meta‐analysis of genome‐wide association data identifies six new risk loci for Parkinson’s disease;Nalls MA;Nat Genet,2014

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5. Mechanisms of lysosomal tubulation and sorting driven by LRRK2;Biochemical Society Transactions;2024-07-31

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