Fibroblast Growth Factor 23 in Feline Chronic Kidney Disease
Author:
Affiliation:
1. Department of Veterinary Clinical Sciences; Royal Veterinary College; Hatfield UK
2. Department of Veterinary Basic Sciences; Royal Veterinary College; London UK
Publisher
Wiley
Subject
General Veterinary
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jvim.12044/fullpdf
Reference39 articles.
1. Feline renal failure: Questions, answers, questions;Lulich;Compend Cont Ed Pract Vet,1992
2. Feline chronic renal failure: Calcium homeostasis in 80 cases diagnosed between 1992 and 1995;Barber;J Small Anim Pract,1998
3. On the pathogenesis of the uremic state. An exposition of the “trade-off hypothesis”;Bricker;N Engl J Med,1972
4. Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23;White;Nat Genet,2000
5. Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia;Yamazaki;J Clin Endocrinol Metab,2002
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