Crizotinib‐induced anti‐cancer activity in human cervical carcinoma cells via ROS ‐dependent mitochondrial depolarization and induction of apoptotic pathway
Author:
Affiliation:
1. Dr. B. R Ambedkar Centre for Biomedical Research University of Delhi, North Campus New Delhi India
Publisher
Wiley
Subject
Obstetrics and Gynaecology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jog.15003
Reference25 articles.
1. Structure Based Drug Design of Crizotinib (PF-02341066), a Potent and Selective Dual Inhibitor of Mesenchymal–Epithelial Transition Factor (c-MET) Kinase and Anaplastic Lymphoma Kinase (ALK)
2. An Orally Available Small-Molecule Inhibitor of c-Met, PF-2341066, Exhibits Cytoreductive Antitumor Efficacy through Antiproliferative and Antiangiogenic Mechanisms
3. Crizotinib: a novel and first-in-class multitargeted tyrosine kinase inhibitor for the treatment of anaplastic lymphoma kinase rearranged nonsmall cell lung cancer and beyond
4. The potential for crizotinib in non-small cell lung cancer: a perspective review
5. FDA Approval Summary: Crizotinib for the Treatment of Metastatic Non-Small Cell Lung Cancer With Anaplastic Lymphoma Kinase Rearrangements
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