Ginsenoside Rg1 improves Alzheimer's disease by regulating oxidative stress, apoptosis, and neuroinflammation through Wnt/GSK‐3β/β‐catenin signaling pathway-Reference-Cited by-同舟云学术

Ginsenoside Rg1 improves Alzheimer's disease by regulating oxidative stress, apoptosis, and neuroinflammation through Wnt/GSK‐3β/β‐catenin signaling pathway

Published:2022-03-31 Issue:6 Volume:99 Page:884-896
ISSN:1747-0277
Container-title:Chemical Biology & Drug Design
language:en
Short-container-title:Chem Biol Drug Des

Author:

Yang Yi¹,Wang Limei¹²,Zhang Caijun³,Guo Yuqian⁴,Li Jintao¹,Wu Chao⁵,Jiao Jianlin⁶,Zheng Hong¹

Affiliation:

1. Department of Laboratory Animal Science Kunming Medical University Kunming China

2. State Key Laboratory for Conservation and Utilization of Bio‐Resources in Yunnan Yunnan University Kunming China

3. Experiment Center of Basic Medical Science Kunming Medical University Kunming China

4. Affiliated Hospital of Medical Sergeant School Army Medical University Shijiazhuang China

5. Department of Pharmacy Hefei Ion Medical Center Hefei China

6. Science and Technology Achievement Incubation Center Kunming Medical University Kunming China

Funder

Applied Basic Research Foundation of Yunnan Province

Publisher

Wiley

Subject

Molecular Medicine,Biochemistry,Drug Discovery,Pharmacology,Organic Chemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cbdd.14041

Reference75 articles.

1. β-catenin is a target for the ubiquitin–proteasome pathway

2. Deciphering the Role of WNT Signaling in Metabolic Syndrome–Linked Alzheimer’s Disease

3. Ginsenoside Rb1 inhibits fibrillation and toxicity of alpha-synuclein and disaggregates preformed fibrils

4. High plasticity of axonal pathology in Alzheimer’s disease mouse models

5. From tau phosphorylation to tau aggregation: what about neuronal death?

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