Ablation of the N terminus of cardiac essential light chain promotes the super‐relaxed state of myosin and counteracts hypercontractility in hypertrophic cardiomyopathy mutant mice
Author:
Affiliation:
1. Department of Molecular and Cellular Pharmacology University of Miami Miller School of Medicine FL USA
2. University of Miami Coral Gables FL USA
Funder
National Heart, Lung, and Blood Institute
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/febs.15243
Reference47 articles.
1. Myosin essential light chain in health and disease
2. The formation and maturation of skeletal muscle in the mouse: the myosin MLC1F/3Fgene as a molecular model
3. Different expressivity of a ventricular essential myosin light chain gene Ala57Gly mutation in familial hypertrophic cardiomyopathy
4. Molecular modeling of the myosin-S1(A1) isoform
5. The Role of the N-Terminus of the Myosin Essential Light Chain in Cardiac Muscle Contraction
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