<scp>2‐Arachidonoylglycerol</scp> ‐mediated endocannabinoid signaling modulates mechanical hypersensitivity associated with alcohol withdrawal in mice-Reference-Cited by-同舟云学术

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2‐Arachidonoylglycerol ‐mediated endocannabinoid signaling modulates mechanical hypersensitivity associated with alcohol withdrawal in mice

Published:2022-10-12 Issue:11 Volume:46 Page:2010-2024
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin & Exp Res

Author:

Morgan Amanda¹,Adank Danielle²,Johnson Keenan¹,Butler Emily³,Patel Sachin¹^ORCID

Affiliation:

1. Department of Psychiatry and Behavioral Sciences Northwestern University Feinberg School of Medicine Chicago Illinois USA

2. Vanderbilt Brain Institute, Vanderbilt University Nashville Tennessee USA

3. Interdisciplinary Program in Neuroscience Vanderbilt University Nashville Tennessee USA

Funder

National Institute on Alcohol Abuse and Alcoholism

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acer.14949

Reference75 articles.

1. Primary Care Patients with Drug Use Report Chronic Pain and Self-Medicate with Alcohol and Other Drugs

2. Involvement of delta opioid receptors in alcohol withdrawal-induced mechanical allodynia in male C57BL/6 mice

3. Gabapentin for Alcohol-Related Disorders

4. Neural mechanisms of pain and alcohol dependence

5. Functional Redundancy Between Canonical Endocannabinoid Signaling Systems in the Modulation of Anxiety

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Neonatal dysregulation of 2-arachidonoylglycerol induces impaired brain function in adult mice;Neuropharmacology;2024-10

2. Alcohol Use Disorder and Chronic Pain: An Overlooked Epidemic;American Journal of Psychiatry;2024-05-01

3. Histone deacetylase inhibitor decreases hyperalgesia in a mouse model of alcohol withdrawal‐induced hyperalgesia;Alcohol, Clinical and Experimental Research;2024-02-20

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