Defects in meristem maintenance, cell division, and cytokinin signaling are early responses in the boron deficient maize mutant <i>tassel‐less1</i>-Reference-Cited by-同舟云学术

Defects in meristem maintenance, cell division, and cytokinin signaling are early responses in the boron deficient maize mutant tassel‐less1

Published:2022-03 Issue:2 Volume:174 Page:
ISSN:0031-9317
Container-title:Physiologia Plantarum
language:en
Short-container-title:Physiologia Plantarum

Author:

Matthes Michaela S.¹^ORCID,Darnell Zoe¹,Best Norman B.¹^ORCID,Guthrie Katy¹^ORCID,Robil Janlo M.¹^ORCID,Amstutz Jen¹,Durbak Amanda²,McSteen Paula¹^ORCID

Affiliation:

1. Division of Biological Sciences Bond Life Sciences Center, Interdisciplinary Plant Group, and Missouri Maize Center, University of Missouri Columbia Missouri USA

2. Division of Biological Sciences University of Missouri Columbia Missouri USA

Funder

National Institute of Food and Agriculture

Publisher

Wiley

Subject

Cell Biology,Plant Science,Genetics,General Medicine,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ppl.13670

Reference105 articles.

1. Boron deficiency results in early repression of a cytokinin receptor gene and abnormal cell differentiation in the apical root meristem of Arabidopsis thaliana

2. Description and mapping of the tassel‐less (tls1) mutation;Albertsen M.C.;Maize Newsletter,1993

3. Andrews S.(2010)FastQC: A quality control tool for high throughput sequence data. Available from:http://www.bioinformatics.babraham.ac.uk/projects/fastqc/

4. Unraveling the KNOTTED1 regulatory network in maize meristems

5. The maize Gα gene COMPACT PLANT2 functions in CLAVATA signalling to control shoot meristem size

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