Mechanism of Acetaminophen-Induced Apoptosis in Cultured Cells: Roles of Caspase-3, DNA Fragmentation Factor, and the Ca2+ and Mg2+ Endonuclease DNAS1L3-Reference-Cited by-同舟云学术

Mechanism of Acetaminophen-Induced Apoptosis in Cultured Cells: Roles of Caspase-3, DNA Fragmentation Factor, and the Ca2+ and Mg2+ Endonuclease DNAS1L3

Published:2008-07-25 Issue:1 Volume:94 Page:19-29
ISSN:1742-7835
Container-title:Basic & Clinical Pharmacology & Toxicology
language:en
Short-container-title:

Author:

Boulares A. Hamid,Ren Tao

Publisher

Wiley

Subject

Pharmacology,Toxicology,General Medicine

Reference48 articles.

1. Acetaminophen inhibits NF-κB activation by interfering with the oxidant signal in murine Hepa 1-6 cells;Boulares;Toxicol. Sci.,2000

2. Regulation of DNAS1L3 endonuclease activity by poly(ADP-ribosyl)ation during etoposide-induced apoptosis. Role of poly(ADP-ribose) polymerase-1 cleavage in endonuclease activation;Boulares;J. Biol. Chem.,2002a

3. Roles of DNA fragmentation Factor and poly(ADP-ribose) polymerase-1 in sensitization of fibroblasts to tumor necrosis factor-induced apoptosis;Boulares;Biochem. Biophys. Res. Commun.,2002b

4. The PARP-1-regulated endonuclease DNAS1L3 is required for etoposide-induced internucleosomal DNA fragmentation and increases etoposide cytotoxicity in transfected osteosarcoma cells;Boulares;Cancer Res.,2002c

5. Acetaminophen induces a caspase-dependent and Bcl-xL sensitive apoptosis in human hepatoma cells and lymphocytes;Boulares;Basic & Clinical Pharmacology & Toxicology,2002d

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