Chronic Alcohol Exposure Enhances Lipopolysaccharide-Induced Lung Injury in Mice: Potential Role of Systemic Tumor Necrosis Factor-Alpha-Reference-Cited by-同舟云学术

Chronic Alcohol Exposure Enhances Lipopolysaccharide-Induced Lung Injury in Mice: Potential Role of Systemic Tumor Necrosis Factor-Alpha

Published:2015-09-18 Issue:10 Volume:39 Page:1978-1988
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Res

Author:

Massey Veronica L.¹²,Poole Lauren G.¹²,Siow Deanna L.¹²,Torres Edilson³,Warner Nikole L.⁴,Schmidt Robin H.¹²,Ritzenthaler Jeffrey D.³,Roman Jesse³,Arteel Gavin E.¹²

Affiliation:

1. Department of Pharmacology and Toxicology; University of Louisville Health Sciences Center; Louisville Kentucky

2. University of Louisville Alcohol Research Center; Louisville Kentucky

3. Division of Pulmonary, Critical Care and Sleep Disorders Medicine; Department of Medicine; University of Louisville Health Sciences Center; Louisville Kentucky

4. Department of Micriobiology and Immunology; University of Louisville Health Sciences Center; Louisville Kentucky

Funder

National Institutes of Health

University of Louisville

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acer.12855/fullpdf

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4. Elimination of macrophages by liposome-encapsulated dichloromethylene diphosphonate suppresses the endotoxin-induced priming of Kupffer cells;Bautista;J Leukoc Biol,1994

5. Chronic ethanol ingestion increases expression of the angiotensin II type 2 (AT2) receptor and enhances tumor necrosis factor-alpha- and angiotensin II-induced cytotoxicity via AT2 signaling in rat alveolar epithelial cells;Bechara;Alcohol Clin Exp Res,2003

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