Endothelial nitric oxide deficiency promotes Alzheimer's disease pathology-Reference-Cited by-同舟云学术

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Endothelial nitric oxide deficiency promotes Alzheimer's disease pathology

Published:2013-06-27 Issue:5 Volume:127 Page:691-700
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Austin Susan A.¹,Santhanam Anantha V.¹,Hinton David J.²,Choi Doo-Sup²,Katusic Zvonimir S.¹²

Affiliation:

1. Department of Anesthesiology; Mayo Clinic; Rochester Minnesota USA

2. Molecular Pharmacology and Experimental Therapeutics; Mayo Clinic; Minnesota USA

Funder

National Institutes of Health

Mayo Alzheimer's Disease Research Center

AHA

Clinical Pharmacology Training Grant

Samuel Johnson Foundation for Genomics of Addiction Program at Mayo Clinic

Mayo Foundation

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12334/fullpdf

Reference51 articles.

1. Transgenic expression of intraneuronal Abeta42 but not Abeta40 leads to cellular Abeta lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology;Abramowski;J. Neurosci.,2012

2. Nitric oxide acts as a retrograde messenger during long-term potentiation in cultured hippocampal neurons;Arancio;J. Physiol. Paris,1996

3. Endothelial nitric oxide modulates expression and processing of amyloid precursor protein;Austin;Circ. Res.,2010

4. Supplementation of nitric oxide attenuates AbetaPP and BACE1 protein in cerebral microcirculation of eNOS-deficient mice;Austin;J. Alzheimers Dis.,2012

5. Nitric oxide inhibits endothelial IL-1[beta]-induced ICAM-1 gene expression at the transcriptional level decreasing Sp1 and AP-1 activity;Berendji-Grun;Mol. Med.,2001

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