Altered hippocampal kynurenine pathway metabolism contributes to hyperexcitability in human mesial temporal lobe epilepsy–hippocampal sclerosis-Reference-Cited by-同舟云学术

Altered hippocampal kynurenine pathway metabolism contributes to hyperexcitability in human mesial temporal lobe epilepsy–hippocampal sclerosis

Published:2021-06-22 Issue:19 Volume:178 Page:3959-3976
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:Br J Pharmacol

Author:

Dey Soumil¹,Banerjee Dixit Aparna²,Tripathi Manjari³,Doddamani Ramesh Sharanappa¹,Sharma Mehar Chand⁴,Lalwani Sanjeev⁵,Chandra Poodipedi Sarat¹,Banerjee Jyotirmoy⁶^ORCID

Affiliation:

1. Department of Neurosurgery All India Institute of Medical Sciences New Delhi India

2. Dr. B. R. Ambedkar Centre for Biomedical Research University of Delhi New Delhi India

3. Department of Neurology All India Institute of Medical Sciences New Delhi India

4. Department of Pathology All India Institute of Medical Sciences New Delhi India

5. Department of Forensic Medicine and Toxicology All India Institute of Medical Sciences New Delhi India

6. Department of Biophysics All India Institute of Medical Sciences New Delhi India

Funder

Ministry of Science and Technology

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bph.15534

Reference54 articles.

1. THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Enzymes

2. THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Ion channels

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4. Functional G-protein-coupled receptor 35 is expressed by neurons in the CA1 field of the hippocampus

5. Kynurenic acid inhibits glutamatergic transmission to CA1 pyramidal neurons via α7 nAChR-dependent and -independent mechanisms

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