MiR-101a ameliorates AngII-mediated hypertensive nephropathy by blockade of TGFβ/Smad3 and NF-κB signalling in a mouse model of hypertension
Author:
Affiliation:
1. Wuxi No. 2 People's Hospital; Affiliated Hospital of Nanjing Medical University; Wuxi China
Publisher
Wiley
Subject
Physiology (medical),Pharmacology,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/1440-1681.13042/fullpdf
Reference27 articles.
1. Pathogenesis of hypertension;Oparil;Ann Intern Med,2003
2. Hypertensive nephropathy. Moving from classic to emerging pathogenetic mechanisms;Seccia;J Hypertens,2017
3. Preliminary study of microRNA-126 as a novel therapeutic target for primary hypertension;Liu;Int J Mol Med,2018
4. The microRNA miR-433 promotes renal fibrosis by amplifying the TGF-beta/Smad3-Azin1 pathway;Li;Kidney Int,2013
5. Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis;Rooij;Proc Natl Acad Sci USA,2008
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