Metabolic remodelling in obesity and type 2 diabetes: pathological or protective mechanisms in response to nutrient excess?-Reference-Cited by-同舟云学术

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Metabolic remodelling in obesity and type 2 diabetes: pathological or protective mechanisms in response to nutrient excess?

Published:2014-12-22 Issue:1 Volume:42 Page:109-115
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Connor Timothy¹,Martin Sheree D¹,Howlett Kirsten F²,McGee Sean L¹³

Affiliation:

1. Metabolic Remodelling Laboratory; Metabolic Research Unit; School of Medicine; Deakin University; Geelong Vic. Australia

2. Centre for Physical Activity and Nutrition; School of Exercise and Nutrition Sciences; Deakin University; Geelong Vic. Australia

3. Division of Cell Signalling and Metabolism; Baker IDI Heart and Diabetes Institute; Melbourne Vic. Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/1440-1681.12315/fullpdf

Reference68 articles.

1. Mechanisms for insulin resistance: Common threads and missing links;Samuel;Cell,2012

2. Insulin resistance in the defense against obesity;Saltiel;Cell Metab.,2012

3. Insulin resistance is a cellular antioxidant defense mechanism;Hoehn;Proc. Natl Acad. Sci. USA,2009

4. Hepatic insulin resistance is sufficient to produce dyslipidemia and susceptibility to atherosclerosis;Biddinger;Cell Metab.,2008

5. A noncanonical, GSK3-independent pathway controls postprandial hepatic glycogen deposition;Wan;Cell Metab.,2013

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