Angiotensin receptor-1A knockout leads to hydronephrosis not associated with a loss of pyeloureteric peristalsis in the mouse renal pelvis
Author:
Affiliation:
1. Department of Physiology; School of Biomedical Sciences; Monash University; Clayton Victoria Australia
2. Department of Cell Physiology; Nagoya City University Graduate School of Medical Sciences; Nagoya Japan
Funder
Grant-in-Aid for Scientific Research (B)
Japan Society for Promotion of the Science
Publisher
Wiley
Subject
Physiology (medical),Pharmacology,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/1440-1681.12560/fullpdf
Reference28 articles.
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2. Role of Ca2+ entry and Ca2+ stores in atypical smooth muscle cell autorhythmicity in the mouse renal pelvis;Lang;Br. J. Pharmacol.,2007
3. Spontaneous electrical and Ca2+ signals in the mouse renal pelvis that drive pyeloureteric peristalsis;Lang;Clin. Exp. Pharmacol. Physiol.,2010
4. Pacemaker activity of the pelvicalyceal border recorded by an intracellular glass microelectrode;Tsuchida;Urol. Int.,1992
5. Spontaneous electrical and Ca2+ signals in typical and atypical smooth muscle cells and interstitial cell of Cajal-like cells of mouse renal pelvis;Lang;J. Physiol.,2007
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