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Activation of imidazoline-I3 receptors ameliorates pancreatic damage

  • Published:2015-08-25 Issue:9 Volume:42 Page:964-971
  • ISSN:0305-1870
  • Container-title:Clinical and Experimental Pharmacology and Physiology
  • language:en
  • Short-container-title:Clin Exp Pharmacol Physiol

Author:

Li Yingxiao1,Cheng Kai-Chun1,Asakawa Akihiro1,Amitani Haruka1,Takimoto Yoshiyuki2,Runtuwene Joshua1,Inui Akio2

Affiliation:

1. Department of Psychosomatic Internal Medicine; Kagoshima University Graduate School of Medical and Dental Sciences; Kagoshima Japan

2. Department of Stress Sciences and Psychosomatic Medicine; Graduate School of Medicine; The University of Tokyo; Tokyo Japan

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Reference39 articles.

1. Islet pathology and the pathogenesis of type 1 and type 2 diabetes mellitus revisited;Klöppel;Surv. Synth. Pathol. Res.,1985

2. β-cell deficit and increased β-cell apoptosis in humans with type 2 diabetes;Butler;Diabetes,2003

3. A choice of death: The signal-transduction of immune-mediated β-cell apoptosis;Eizirik;Diabetologia,2001

4. beta-cell apoptosis: Stimuli and signaling;Mandrup-Poulsen;Diabetes,2001

5. Mechanisms of pancreatic beta-cell death in type 1 and type 2 diabetes: Many differences, few similarities;Cnop;Diabetes,2005

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