Activation of PKCα participates in the reduction of Ikur in atrial myocytes induced by tumour necrosis factor‐α-Reference-Cited by-同舟云学术

Activation of PKCα participates in the reduction of Ikur in atrial myocytes induced by tumour necrosis factor‐α

Published:2020-10-09 Issue:3 Volume:48 Page:435-442
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Zhou Hui‐shan¹²,Peng De‐wei¹³,Lai Ying‐yu¹³,Li Qian¹³,Zhao Jun‐fei¹,Deng Chun‐yu¹³,Yang Hui¹³^ORCID,Li Teng⁴,Wang Zhao‐yu¹³,Xu Yu‐wen¹³,Xue Yu‐mei¹³,Wu Shu‐lin¹³^ORCID,Guo Hui‐ming¹,Rao Fang¹³^ORCID

Affiliation:

1. Guangdong Cardiovascular Institute Guangdong Provincial People's Hospital Guangdong Academy of Medical Sciences Guangzhou China

2. Panyu Central Hospital Guangzhou China

3. Guangdong Provincial Key Laboratory of Clinical Pharmacology Research Center of Medical Sciences Guangdong Provincial People's Hospital Guangzhou China

4. Arrhythmia Department Fuwai Hospital Chinese Academy of Medical Sciences Shenzhen China

Funder

Guangzhou Municipal Science and Technology Project

National Natural Science Foundation of China

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1440-1681.13407

Reference26 articles.

1. Outward K + Current Densities and Kv1.5 Expression Are Reduced in Chronic Human Atrial Fibrillation

2. Pharmacology of voltage-gated potassium channel Kv1.5—impact on cardiac excitability

3. Potassium currents in the heart: functional roles in repolarization, arrhythmia and therapeutics

4. In Humans, Chronic Atrial Fibrillation Decreases the Transient Outward Current and Ultrarapid Component of the Delayed Rectifier Current Differentially on Each Atria and Increases the Slow Component of the Delayed Rectifier Current in Both

5. Pathology-specific effects of the I Kur /I to /I K,ACh blocker AVE0118 on ion channels in human chronic atrial fibrillation

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