Age-dependent electrocardiographic changes in Pgc-1β deficient murine hearts-Reference-Cited by-同舟云学术

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Age-dependent electrocardiographic changes in Pgc-1β deficient murine hearts

Published:2017-11-29 Issue:2 Volume:45 Page:174-186
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Ahmad Shiraz¹,Valli Haseeb¹,Salvage Samantha C²,Grace Andrew A²,Jeevaratnam Kamalan¹³⁴^ORCID,Huang Christopher L-H¹²^ORCID

Affiliation:

1. Physiological Laboratory; University of Cambridge; Cambridge United Kingdom

2. Department of Biochemistry; University of Cambridge; Cambridge United Kingdom

3. Faculty of Health and Medical Sciences; University of Surrey; Guildford Surrey United Kingdom

4. PU-RCSI School of Medicine; Perdana University; Selangor Darul Ehsan Malaysia

Funder

Wellcome Trust

Medical Research Council

British Heart Foundation

Sudden Arrhythmic Death Syndrome (SADS) UK

Fundamental Research Grant Scheme, Ministry of Education, Malaysia

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/1440-1681.12863/fullpdf

Reference52 articles.

1. Epidemiology and genetics of sudden cardiac death;Deo;Circulation,2012

2. Epidemiology of atrial fibrillation: European perspective;Zoni-Berisso;Clin Epidemiol,2014

3. Diabetes and the risk of sudden cardiac death, the Atherosclerosis Risk in Communities study;Kucharska-Newton;Acta Diabetol,2010

4. Oxidative damage to mitochondrial DNA in atrial muscle of patients with atrial fibrillation;Lin;Free Radic Biol Med,2003

5. Mitochondrial dysfunction and redox signaling in atrial tachyarrhythmia;Bukowska;Exp Biol Med,2008

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3. Cardiomyocyte electrophysiology and its modulation: current views and future prospects;Philosophical Transactions of the Royal Society B: Biological Sciences;2023-05

4. Molecular basis of ventricular arrhythmogenicity in a Pgc-1α deficient murine model;Molecular Genetics and Metabolism Reports;2021-06

5. Mitochondrial Dysfunction Increases Arrhythmic Triggers and Substrates; Potential Anti-arrhythmic Pharmacological Targets;Frontiers in Cardiovascular Medicine;2021-02-15

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