Tenacissoside H promotes neurological recovery of cerebral ischaemia/reperfusion injury in mice by modulating inflammation and oxidative stress via TrkB pathway-Reference-Cited by-同舟云学术

Tenacissoside H promotes neurological recovery of cerebral ischaemia/reperfusion injury in mice by modulating inflammation and oxidative stress via TrkB pathway

Published:2021-03 Issue:5 Volume:48 Page:757-769
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Zhang Rui¹,Liu Cui²,Li Yang¹,Chen Liang³,Xiang Jianfeng³^ORCID

Affiliation:

1. Department of NICU Affiliated Hospital of Qingdao University Qingdao China

2. Department of Cardiovascular Surgery Affiliated Hospital of Qingdao University Qingdao China

3. Interventional Department Shanghai Jiao Tong University Affiliated Sixth People's Hospital South Campus (Shanghai Fengxian District Central Hospital) Shanghai China

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1440-1681.13398

Reference50 articles.

1. Remodeling of Cerebral Microcirculation after Ischemia-Reperfusion

2. Blood−brain barrier breakdown and neovascularization processes after stroke and traumatic brain injury

3. Bone marrow mesenchymal stromal cells alleviate brain white matter injury via the enhanced proliferation of oligodendrocyte progenitor cells in focal cerebral ischemic rats

4. Current Mechanistic Concepts in Ischemia and Reperfusion Injury

5. ROS-Mediated NLRP3 Inflammasome Activation in Brain, Heart, Kidney, and Testis Ischemia/Reperfusion Injury

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