Increased inward rectifier K + current of coronary artery smooth muscle cells in spontaneously hypertensive rats; partial compensation of the attenuated endothelium‐dependent relaxation via Ca 2+ ‐activated K + channels-Reference-Cited by-同舟云学术

Increased inward rectifier K + current of coronary artery smooth muscle cells in spontaneously hypertensive rats; partial compensation of the attenuated endothelium‐dependent relaxation via Ca 2+ ‐activated K + channels

Published:2019-09-15 Issue:1 Volume:47 Page:38-48
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Kim Hae Jin¹²,Yin Ming Zhe¹²,Cho Suhan¹²,Kim Sung Eun¹²,Choi Seong Woo¹³,Ye Sang Kyu²³⁴,Yoo Hae Young⁵,Kim Sung Joon¹²³^ORCID

Affiliation:

1. Department of Physiology Seoul National University College of Medicine Seoul Korea

2. Department of Biomedical Sciences Seoul National University College of Medicine Seoul Korea

3. Ischemic/Hypoxic Disease Institute Seoul National University College of Medicine Seoul Korea

4. Department of Pharmacology Seoul National University College of Medicine Seoul Korea

5. Chung‐Ang University Red Cross College of Nursing Seoul Korea

Funder

National Research Foundation of Korea

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1440-1681.13168

Reference34 articles.

1. An endothelium-derived hyperpolarizing factor distinct from NO and prostacyclin is a major endothelium-dependent vasodilator in resistance vessels of wild-type and endothelial NO synthase knockout mice

2. Connexins and gap junctions in the EDHF phenomenon and conducted vasomotor responses

3. TRPV4 calcium entry channel: a paradigm for gating diversity

4. Role of arachidonic acid lipoxygenase metabolites in the regulation of vascular tone

5. Hydrogen peroxide as an endothelium-derived hyperpolarizing factor

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