Knockout of cardiac troponin <scp>I‐interacting</scp> kinase leads to cardiac dysfunction and remodelling-Reference-Cited by-同舟云学术

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Knockout of cardiac troponin I‐interacting kinase leads to cardiac dysfunction and remodelling

Published:2022-07-22 Issue:11 Volume:49 Page:1169-1178
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharma Physio

Author:

Qu Huilin¹,Zhang Yue¹,Zhang Wei¹,Zhu Yaxin¹,Xu Ruixia¹

Affiliation:

1. State Key Laboratory of Cardiovascular Disease, National Center for Cardiovascular Disease, Fu Wai Hospital Chinese Academy of Medical Sciences and Peking Union Medical College Beijing China

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1440-1681.13699

Reference31 articles.

1. Cloning and characterization of a novel cardiac-specific kinase that interacts specifically with cardiac troponin I

2. Inhibition of the Cardiomyocyte-Specific Kinase TNNI3K Limits Oxidative Stress, Injury, and Adverse Remodeling in the Ischemic Heart

3. AOP-1 interacts with cardiac-specific protein kinase TNNI3K and down-regulates its kinase activity

4. The ankyrin repeat as molecular architecture for protein recognition

5. Identification of the dual specificity and the functional domains of the cardiac‐specific protein kinase TNNI3K;Feng Y;Gen Physiol Biophys,2007

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