Cytochrome P4502E1 inhibitor, a potential oxidative stress regulator in liver diseases
Author:
Affiliation:
1. School of Pharmacy; Anhui Key Laboratory of Bioactivity of Natural Products
2. Institute for Liver Diseases of Anhui Medical University; Anhui Medical University; Hefei China
Publisher
Wiley
Subject
Infectious Diseases,Hepatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/hepr.12160/fullpdf
Reference10 articles.
1. Cytochrome P450 CYP2E1, but not nicotinamide adenine dinucleotide phosphate oxidase, is required for ethanol-induced oxidative DNA damage in rodent liver;Bradford;Hepatology,2005
2. Chronic alcohol-induced liver injury and oxidant stress are decreased in cytochrome P4502E1 knockout mice and restored in humanized cytochrome P4502E1 knock-in mice;Lu;Free Radic Biol Med,2010
3. Cytochrome P4502E1 inhibitor, chlormethiazole, decreases lipopolysaccharide-induced inflammation in rat Kupffer cells with ethanol treatment;Ye;Hepatol Res,2013
4. Regulation of cytochrome P450 2e1 expression by ethanol: role of oxidative stress-mediated pkc/jnk/sp1 pathway;Jin;Cell Death Dis,2013
5. Susceptibility of L-FABP-/- mice to oxidative stress in early-stage alcoholic liver;Smathers;J Lipid Res,2013
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