Different signalling in infarcted and non‐infarcted areas of rat failing hearts: A role of necroptosis and inflammation
Author:
Affiliation:
1. Faculty of Pharmacy, Department of Pharmacology and Toxicology Comenius University in Bratislava Bratislava Slovak Republic
2. Institute of Physiology of the Czech Academy of Sciences Prague Czech Republic
Funder
Vedecká Grantová Agentúra MŠVVaŠ SR a SAV
Agentúra na Podporu Výskumu a Vývoja
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14536
Reference64 articles.
1. Cardiomyocyte Cell Cycle Regulation
2. RIP3, a kinase promoting necroptotic cell death, mediates adverse remodelling after myocardial infarction
3. CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis
4. Transforming Growth Factor β–Activated Kinase 1 Signaling Pathway Critically Regulates Myocardial Survival and Remodeling
5. Analysis of necroptotic proteins in failing human hearts
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