AraC‐type regulators HilC and RtsA are directly controlled by an intestinal fatty acid to regulate Salmonella invasion
Author:
Affiliation:
1. Department of Population Medicine and Diagnostic Sciences College of Veterinary Medicine Cornell University Ithaca New York USA
2. Department of Molecular Medicine Cornell University Ithaca New York USA
Funder
National Institute of Allergy and Infectious Diseases
Publisher
Wiley
Subject
Molecular Biology,Microbiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/mmi.14835
Reference60 articles.
1. Self-destructive cooperation mediated by phenotypic noise
2. Characterization of two novel regulatory genes affecting Salmonella invasion gene expression
3. Diffusible signal factor signaling regulates multiple functions in the opportunistic pathogen Stenotrophomonas maltophilia
4. HilE Interacts with HilD and Negatively Regulates hilA Transcription and Expression of the Salmonella enterica Serovar Typhimurium Invasive Phenotype
5. The fimYZ Genes Regulate Salmonella enterica Serovar Typhimurium Invasion in Addition to Type 1 Fimbrial Expression and Bacterial Motility
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1. HilE represses the activity of the Salmonella virulence regulator HilD via a mechanism distinct from that of intestinal long-chain fatty acids;Journal of Biological Chemistry;2023-12
2. Shigella flexneri utilizes intestinal signals to control its virulence;Gut Microbes;2023-09-23
3. Anti-infective activities of long-chain fatty acids against foodborne pathogens;FEMS Microbiology Reviews;2023-07
4. Recombinant production of a diffusible signal factor inhibits Salmonella invasion and animal carriage;Gut Microbes;2023-05-09
5. Salmonella Invasion Is Controlled by Competition among Intestinal Chemical Signals;mBio;2023-04-25
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