The Presence and involvement of interleukin‐17 in apical periodontitis-Reference-Cited by-同舟云学术

The Presence and involvement of interleukin‐17 in apical periodontitis

Published:2019-03-27 Issue: Volume: Page:
ISSN:0143-2885
Container-title:International Endodontic Journal
language:en
Short-container-title:Int Endod J

Author:

Xiong H.¹^ORCID,Wei L.¹,Peng B.¹^ORCID

Affiliation:

1. The State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei‐MOST) & Key Laboratory of Oral Biomedicine Ministry of Education School & Hospital of Stomatology Wuhan University Wuhan China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

General Dentistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/iej.13112

Reference62 articles.

1. Immunoexpression of Interleukin 17 in Apical Periodontitis Lesions

2. IL-17 Receptor A Signaling Is Protective in Infection-Stimulated Periapical Bone Destruction

3. Immunoexpression of Interleukin 17, Transforming Growth Factor β1, and Forkhead Box P3 in Periapical Granulomas, Radicular Cysts, and Residual Radicular Cysts

4. Simultaneous analysis of T helper subsets (Th1, Th2, Th9, Th17, Th22, Tfh, Tr1 and Tregs) markers expression in periapical lesions reveals multiple cytokine clusters accountable for lesions activity and inactivity status

5. The role of IL-6 on apical periodontitis: a systematic review

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