Cellular origins and molecular mechanisms of Barrett's esophagus and esophageal adenocarcinoma

Author:

Fang Yu12,Chen Xiaoxin13,Bajpai Manisha4,Verma Amit4,Das Kiron M.4,Souza Rhonda F.5,Garman Katherine S.6,Donohoe Claire L.7,O'Farrell Naoimh J.7,Reynolds John V.7,Dvorak Katerina8

Affiliation:

1. Cancer Research Program, JLC-BBRI; North Carolina Central University; Durham North Carolina

2. Department of Cardiovascular and Thoracic Surgery; The Second Xiangya Hospital, Central South University; Changsha China

3. Division of Gastroenterology and Hepatology; Department of Medicine, Center for Esophageal Disease and Swallowing; University of North Carolina at Chapel Hill; Chapel Hill North Carolina

4. Department of Medicine, UMDNJ-RWJMS; Medicine, New Brunswick; New Jersey

5. Department of Medicine; University of Texas Southwestern Medical Center and the VA North Texas Health Care System; Dallas Texas

6. Department of Medicine; Duke University; Durham North Carolina

7. Trinity College Dublin/St. James' Hospital; Trinity Centre for Health Sciences; St. James’ Hospital; Dublin Ireland

8. Department of Cellular and Molecular Medicine; Arizona Cancer Center; University of Arizona; Tucson Arizona

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Reference60 articles.

1. Immunohistochemical study of p53, c-erbB-2, and PCNA in Barrett's esophagus with dysplasia and adenocarcinoma arising from experimental acid or alkaline reflux model;Kawaura;J. Gastroenterol.,2001

2. Review: experimental models for Barrett's esophagus and esophageal adenocarcinoma;Garman;Am. J. Physiol. Gastrointest. Liver Physiol.,2012

3. Gastroesophageal reflux leads to esophageal cancer in a surgical model with mice;Hao;BMC Gastroenterol.,2009

4. Trans-differentiation of a duodenal phenotype on duodenal transplantation of different normal tissues in F344 rats;Watanabe;Hiroshima J. Med. Sci.,2011

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