The clinically used PARP inhibitor olaparib improves organ function, suppresses inflammatory responses and accelerates wound healing in a murine model of third-degree burn injury-Reference-Cited by-同舟云学术

The clinically used PARP inhibitor olaparib improves organ function, suppresses inflammatory responses and accelerates wound healing in a murine model of third-degree burn injury

Published:2017-03-05 Issue:2 Volume:175 Page:232-245
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:British Journal of Pharmacology

Author:

Ahmad Akbar¹,Olah Gabor¹,Herndon David N²³,Szabo Csaba¹³^ORCID

Affiliation:

1. Department of Anesthesiology; The University of Texas Medical Branch; Galveston TX USA

2. Department of Surgery; The University of Texas Medical Branch; Galveston TX USA

3. Shriners Hospital for Children; Galveston TX USA

Funder

Shriners of North America

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13735/fullpdf

Reference107 articles.

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3. Both the H2S biosynthesis inhibitor aminooxyacetic acid and the mitochondrially targeted H2S donor AP39 exert protective effects in a mouse model of burn injury;Ahmad;Pharmacol Res,2016

4. NAD+ depletion is necessary and sufficient for poly(ADP-ribose) polymerase-1-mediated neuronal death;Alano;J Neurosci,2010

5. The Concise Guide to PHARMACOLOGY 2015/16: Overview;Alexander;Brit J Pharmacol,2015

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