Monoamine variability in the chronic model of atypical absence seizures
Author:
Publisher
Wiley
Subject
Neurology (clinical),Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1528-1167.2008.01880.x/fullpdf
Reference44 articles.
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2. Dual control of dorsal raphe serotonergic neurons by GABA(B) receptors. Electrophysiological and microdialysis studies;Abellán;Synapse,2000
3. Simultaneous measurement of dopamine, serotonin, their metabolites and tryptophan in mouse brain homogenates by high-performance liquid chromatography with dual coulometric detection;Alvarez;Biomed Chromatogr,1999
4. Serotonin depletion attenuates AY-9944-mediated atypical absence seizures;Bercovici;Epilepsia,2006
5. 5-HT2 modulation of AY-9944 induced atypical absence seizures;Bercovici;Neurosci Lett,2007
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1. Atypical absence seizures and gene variants: A gene-based review of etiology, electro-clinical features, and associated epilepsy syndrome;Epilepsy & Behavior;2024-02
2. Altered expression of vesicular monoamine transporter 2 in epileptic patients and experimental rats;Synapse;2013-04-18
3. Social deficits in the AY-9944 mouse model of atypical absence epilepsy;Behavioural Brain Research;2013-01
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