Mitochondrial function and pathology in status epilepticus
Author:
Publisher
Wiley
Subject
Neurology (clinical),Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1528-1167.2011.03223.x/fullpdf
Reference5 articles.
1. POLG1 mutations cause a syndromic epilepsy with occipital lobe predilection;Engelsen;Brain,2008
2. A new mtDNA mutation associated with mitochondrial myopathy, encephalomyopathy, lactic acidosis and stroke-like episodes (MELAS);Goto;Biochim Biophys Acta,1991
3. Slowly progressive spread of the stroke-like lesions in MELAS;Iizuka;Neurology,2003
4. Serial diffusion imaging in a case of mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes;Tzoulis;Stroke,2009
5. The spectrum of clinical disease caused by the A467T and W748S POLG mutations: a study of 26 cases;Tzoulis;Brain,2006
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2. What is the role of next generation sequencing in status epilepticus?;Epilepsy & Behavior;2019-12
3. Febrile infection-related epilepsy syndrome (FIRES): prevalence, impact and management strategies;Neuropsychiatric Disease and Treatment;2019-07
Febrile infection-related epilepsy syndrome (FIRES): prevalence, impact and management strategies
4. Retinoencephalopathy with occipital lobe epilepsy in an OPA-1 mutation carrier;Seizure;2019-03
5. Mitochondrial diseases and status epilepticus;Epilepsia;2018-08-29
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