Neonatal Epilepsy Syndromes and GEFS+: Mechanistic Considerations
Author:
Publisher
Wiley
Subject
Clinical Neurology,Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1528-1167.2005.00359.x/fullpdf
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1. Sodium Channel β Subunits: Anything but Auxiliary
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3. A deletion in SCN1B is associated with febrile seizures and early-onset absence epilepsy
4. Mice Lacking Sodium Channel 1 Subunits Display Defects in Neuronal Excitability, Sodium Channel Expression, and Nodal Architecture
5. Reduced sodium channel density, altered voltage dependence of inactivation, and increased susceptibility to seizures in mice lacking sodium channel 2-subunits
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