Pharmacologic Evidence for Abnormal Thalamocortical Functioning in GABAA Receptor beta3 Subunit-Deficient Mice, a Model of Angelman Syndrome
Author:
Publisher
Wiley
Subject
Neurology (clinical),Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1528-1167.2005.00287.x/fullpdf
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1. Angelman syndrome: Consensus for diagnostic criteria
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4. Mutation of the Angelman Ubiquitin Ligase in Mice Causes Increased Cytoplasmic p53 and Deficits of Contextual Learning and Long-Term Potentiation
5. Enhanced Learning and Memory and Altered GABAergic Synaptic Transmission in Mice Lacking the α5 Subunit of the GABAAReceptor
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