Molecular basis of self-sustaining seizures and pharmacoresistance during status epilepticus: The receptor trafficking hypothesis revisited-Reference-Cited by-同舟云学术

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Molecular basis of self-sustaining seizures and pharmacoresistance during status epilepticus: The receptor trafficking hypothesis revisited

Published:2009-12 Issue: Volume:50 Page:16-18
ISSN:0013-9580
Container-title:Epilepsia
language:en
Short-container-title:

Author:

Wasterlain Claude G.,Liu Hantao,Naylor David E.,Thompson Kerry W.,Suchomelova Lucy,Niquet Jerome,Mazarati Audrey M.,Baldwin Roger A.

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1528-1167.2009.02375.x/fullpdf

Reference10 articles.

1. Status epilepticus increases the intracellular accumulation of GABAA receptors;Goodkin;J Neurosci,2005

2. Subunit-specific trafficking of GABA(A) receptors during status epilepticus;Goodkin;J Neurosci,2008

3. Rapid seizure-induced reduction of benzodiazepine and Zn2 sensitivity of hippocampal dentate granule cell GABAA receptors;Kapur;J Neurosci,1997

4. Mechanisms of drug resistance in status epilepticus;L��scher;Epilepsia,2007

5. Self-sustaining status epilepticus after brief electrical stimulation of the perforant path;Mazarati;Brain Res,1998

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