Tris DBA ameliorates IgA nephropathy by blunting the activating signal of NLRP3 inflammasome through SIRT1‐ and SIRT3‐mediated autophagy induction-Reference-Cited by-同舟云学术

Tris DBA ameliorates IgA nephropathy by blunting the activating signal of NLRP3 inflammasome through SIRT1‐ and SIRT3‐mediated autophagy induction

Published:2020-11 Issue:23 Volume:24 Page:13609-13622
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Wu Chung‐Yao¹,Hua Kuo‐Feng²,Yang Shin‐Ruen¹,Tsai Yi‐Shan¹,Yang Shun‐Min³,Hsieh Chih‐Yu⁴⁵,Wu Chia‐Chao⁶,Chang Jia‐Feng⁴⁵,Arbiser Jack L.⁷⁸,Chang Chiz‐Tzung⁹,Chen Ann¹³^ORCID,Ka Shuk‐Man¹⁰

Affiliation:

1. Graduate Institute of Life Sciences National Defense Medical Center Taipei Taiwan

2. Department of Biotechnology and Animal Science National Ilan University Ilan Taiwan

3. Department of Pathology Tri‐Service General Hospital National Defense Medical Center Taipei Taiwan

4. Department of Internal Medicine En Chu Kong Hospital New Taipei City Taiwan

5. Renal Care Joint Foundation New Taipei City Taiwan

6. Division of Nephrology Department of Internal Medicine Tri‐Service General Hospital National Defense Medical Center Taipei Taiwan

7. Department of Dermatology Emory School of Medicineand Winship Cancer Institute Atlanta GA USA

8. Atlanta Veterans Administration Medical Center Decatur GA USA

9. Division of Nephrology Department of Internal Medicine China Medical University Hospital Taichung Taiwan

10. Graduate Institute of Aerospace and Undersea Medicine Department of Medicine National Defense Medical Center Taipei Taiwan

Funder

Ministry of Science and Technology, Taiwan

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.15663

Reference50 articles.

1. Long-Term Results of a Randomized Controlled Trial in Childhood IgA Nephropathy

2. Decoy receptor 3 inhibits renal mononuclear leukocyte infiltration and apoptosis and prevents progression of IgA nephropathy in mice

3. Clinical Features and Outcomes of IgA Nephropathy with Nephrotic Syndrome

4. Antroquinonol mitigates an accelerated and progressive IgA nephropathy model in mice by activating the Nrf2 pathway and inhibiting T cells and NLRP3 inflammasome

5. IgA Nephropathy with Pathologic Features of Membranoproliferative Glomerulonephritis following Burn Injury

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