Defects in MAP 1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed-Reference-Cited by-同舟云学术

Defects in MAP 1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed

Published:2016-01-10 Issue:2 Volume:15 Page:370-379
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Li Wenjiao¹,Zou Jing¹²³,Yue Fei¹,Song Kun¹,Chen Qi¹,McKeehan Wallace L.¹,Wang Fen¹,Xu Guibin¹,Huang Hai¹⁴,Yi Jinglin³,Liu Leyuan¹³⁵

Affiliation:

1. Center for Translational Cancer Research Institute of Biosciences and Technology Texas A&M Health Science Center 2121 W. Holcombe Blvd. Houston TX 77030 USA

2. Department of ophthalmology Xiangya Hospital Central South University Changsha Hunan 410008 China

3. Jiangxi Research Institute of Ophthalmology and Visual Sciences The Affiliated Eye Hospital of Nanchang University Nanchang 330006 China

4. Department of Urology The Sun Yat‐sen Memorial Hospital Sun Yat‐sen University Guangzhou 510120 China

5. Department of Molecular and Cellular Medicine College of Medicine Texas A&M Health Science Center College Station TX 77843 USA

Funder

National Cancer Institute

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12441

Reference46 articles.

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2. Cellular fibronectin stimulates hepatocytes to produce factors that promote alcohol-induced liver injury

3. Developmental expression of LC3α and β: Absence of fibronectin or autophagy phenotype in LC3β knockout mice

4. Targeting the p27 E3 ligase SCFSkp2 results in p27- and Skp2-mediated cell-cycle arrest and activation of autophagy

5. Priming and potentiation of DNA damage response by fibronectin in human colon cancer cells and tumor‐derived myofibroblasts;Wever O;Int. J. Oncol.,2011

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