Pancreatic β-cell dysfunction, expression of iNOS and the effect of phosphodiesterase inhibitors in human pancreatic islets of type 2 diabetes
Author:
Affiliation:
1. Department of Clinical Science, Division of Endocrine Pharmacology; SUS, Malmö, University of Lund; Malmö; Sweden
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1463-1326.2012.01632.x/fullpdf
Reference39 articles.
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2. Glucose generates coincident insulin and somatostatin pulses and antisynchronous glucagon pulses from human pancreatic islets;Hellman;Endocrinology,2009
3. Impaired beta-cell functions induced by chronic exposure of cultured human pancreatic islets to high glucose;Marshak;Diabetes,1999
4. Molecular and metabolic mechanisms of insulin resistance and [beta]-cell failure in type 2 diabetes;Muoio;Nat Rev Mol Cell Biol,2008
5. Glucolipotoxicity: fuel excess and β-cell dysfunction;Poitout;Endocr Rev,2008
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